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The “nausea” effect of erythromycin has been attributed to its excitation of the motilin receptor for decades.  Where’s the research targeting GI motility and motilin?  I’m puzzled.  You’d think it was a money-maker.

Several lines of evidence suggest that motilin and ghrelin are involved in the control of gastrointestinal motility by the activation of receptors on enteric neurons.  Ref.

Yet, it’s only good old erythromycin available for motility.

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